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Revumenib

Deterministic view of the source YAML entity. Clinical authority remains with the cited source IDs and reviewer sign-off state.

IDDRUG-REVUMENIB
TypeDrug
Aliases
RevuforjРевуменіб
Statusreviewed 2026-04-30 | pending_clinical_signoff
DiseasesDIS-AML
SourcesSRC-NCCN-AML-2025

Drug Facts

ClassSelective menin-MLL (KMT2A) interaction inhibitor
MechanismSmall-molecule inhibitor of the menin-KMT2A (MLL) protein-protein interaction. KMT2A-rearranged and NPM1-mutated leukemias are dependent on menin recruitment to KMT2A-fusion or wild-type KMT2A complexes that drive aberrant HOXA / MEIS1 transcription. Disrupting menin-MLL displaces the leukemogenic transcriptional program, inducing differentiation and apoptosis of leukemic blasts. FDA-approved Nov 2024 on the basis of AUGMENT-101 phase 1/2 trial.
Typical dosing163 mg PO q12h with strong CYP3A inhibitor (cobicistat) co-administration (label-required pharmacokinetic boost), OR 270 mg PO q12h without CYP3A inhibitor. Continuous, until disease progression or unacceptable toxicity. Take with or without food. AUGMENT-101 schedule.
Ukraine registeredFalse
NSZU reimbursedFalse
Ukraine last verified2026-04-30

Warnings

Notes

Pivotal trial: AUGMENT-101 (Issa et al., Nature 2024 / NEJM 2025) — phase 1/2 of revumenib monotherapy in R/R KMT2A-r and NPM1-mut acute leukemia. KMT2A-r AML cohort: ORR ~63%, CR/CRh ~23%; durable responses with bridge-to-alloHCT pathway. FDA accelerated approval Nov 2024 for KMT2A-r R/R acute leukemia (≥1 prior line). NPM1-mut cohort under priority review. Differentiation syndrome (~16%) is the signature AE — mandate corticosteroid hold + supportive care; rechallenge after resolution. QTc prolongation requires baseline + serial ECG with electrolyte correction. Ukraine: NOT registered, NOT reimbursed — major access barrier; international referral or EAP / compassionate-use only. Trial source SRC-AUGMENT-101 pending — citing umbrella SRC-NCCN-AML-2025.

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