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Inavolisib

Deterministic view of the source YAML entity. Clinical authority remains with the cited source IDs and reviewer sign-off state.

IDDRUG-INAVOLISIB
TypeDrug
Aliases
ItovebiІναволісіб
Statuspending_clinical_signoff
DiseasesDIS-BREAST
SourcesSRC-ESMO-BREAST-METASTATIC-2024 SRC-NCCN-BREAST-2025

Drug Facts

ClassPI3K-alpha-selective inhibitor (mutant-selective; degrades mutant p110α)
MechanismOrally bioavailable PI3Kα inhibitor that selectively binds and promotes degradation of mutant p110α (PIK3CA-mut) over wild-type — improved tumor selectivity vs alpelisib. Combined with palbociclib + fulvestrant per INAVO120 in PIK3CA-mutated, HR+/HER2-, endocrine-resistant metastatic breast cancer.
Typical dosing9 mg PO once daily, with palbociclib 125 mg PO QD (3 weeks on / 1 week off) + fulvestrant 500 mg IM (standard schedule), until disease progression or unacceptable toxicity.
Ukraine registeredFalse
NSZU reimbursedFalse
Ukraine last verified2026-04-29

Notes

INAVO120 (NCT04191499): Phase III, PIK3CA-mut HR+/HER2- metastatic breast cancer with disease recurrence on/after adjuvant endocrine therapy. Inavolisib + palbociclib + fulvestrant vs placebo + palbociclib + fulvestrant. Median PFS 15.0 vs 7.3 mo (HR 0.43). FDA approval Oct 2024. Pre-treatment PIK3CA testing (tissue or ctDNA) required. Mutant-selective design rationale = lower hyperglycemia + rash burden than alpelisib while preserving efficacy. Trial-source SRC for INAVO120 not yet ingested into KB — FLAG for follow-up.

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