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Gilteritinib

Deterministic view of the source YAML entity. Clinical authority remains with the cited source IDs and reviewer sign-off state.

IDDRUG-GILTERITINIB
TypeDrug
Aliases
XospataГілтеритиніб
Statusreviewed 2026-04-25 | pending_clinical_signoff
DiseasesDIS-AML
SourcesSRC-ELN-AML-2022 SRC-NCCN-AML-2025

Drug Facts

ClassSelective FLT3 + AXL tyrosine kinase inhibitor (Type I)
MechanismType-I FLT3 inhibitor active against both FLT3-ITD and FLT3-TKD (D835) mutations, including activation-loop mutations that confer resistance to Type-II FLT3 inhibitors (quizartinib, sorafenib). Also inhibits AXL (proposed FLT3-TKI resistance mechanism). Approved on the basis of ADMIRAL trial (single-agent vs salvage chemotherapy in R/R FLT3-mutated AML).
Typical dosing120 mg PO once daily, continuous, until disease progression or unacceptable toxicity. Dose modifications for QTc prolongation, differentiation syndrome, posterior reversible encephalopathy syndrome (PRES), pancreatitis. Take with or without food.
Ukraine registeredFalse
NSZU reimbursedFalse
Ukraine last verified2026-04-27

Warnings

Notes

Pivotal trial: ADMIRAL (Perl et al., NEJM 2019) — single-agent gilteritinib vs investigator-choice salvage chemo in R/R FLT3-mutated AML; median OS 9.3 vs 5.6 mo (HR 0.64). Standard 2L+ for R/R FLT3-mutated AML. Type-I selectivity covers both FLT3-ITD and FLT3-TKD/D835 mutations (advantage vs Type-II quizartinib which misses TKD). Ukraine: NOT registered, NOT reimbursed — major access barrier; international referral or EAP Astellas Ukraine compassionate-use pathway required. Differentiation syndrome (3-5%) is the signature AE — mandate corticosteroid hold + supportive care; rechallenge after resolution.

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