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TP53 R282W partial loss-of-function hotspot

Deterministic view of the source YAML entity. Clinical authority remains with the cited source IDs and reviewer sign-off state.

IDBIO-TP53-R282W
TypeBiomarker
Aliases
TP53 R282W partial loss-of-function hotspot mutationTP53 R282W — hotspot з частковою втратою функціїTP53 p.R282Wp53 R282W
Statusreviewed 2026-04-27 | pending_clinical_signoff
DiseasesNone declared
SourcesSRC-NCCN-AML-2025 SRC-NCCN-BCELL-2025 SRC-NCCN-MM-2025

Biomarker Facts

Biomarker typegene_mutation
Mutation details{"exon": "8", "functional_impact": "partial loss_of_function (residual transactivation activity on some targets) + gain-of-function on others", "gene": "TP53", "gene_hugo_id": "11998", "hgvs_coding": "c.844C>T", "hgvs_protein": "p.R282W", "variant_type": "missense"}
Measurement
MethodTumor-tissue NGS panel (TP53 hotspot or full-gene); FFPE acceptable; ctDNA NGS for liquid biopsy
Unitscategorical (positive | negative); VAF % for clonality assessment
Related biomarkersBIO-TP53-MUTATION BIO-TP53-R175H BIO-TP53-R248Q BIO-TP53-R273H

Notes

R282 is the sixth most-mutated TP53 codon, often grouped with R248 and R273 as the canonical hotspot ring. Hybrid structural/contact mutant: R282 stabilizes the L1/S3 loop region adjacent to the DNA-binding surface, and R→W partially destabilizes the fold while altering contact geometry. Functional studies show R282W retains partial transactivation activity on a subset of canonical p53 targets (CDKN1A/p21, BBC3/PUMA at reduced levels) — termed "partial LoF" — while still gaining oncogenic activities. Clinically R282W behaves similarly to other hotspots: in AML/MDS poor chemo response → routes to venetoclax-azacitidine + alloSCT consideration; in CLL/MCL/MM high-risk regimen routing. Distinction from full LoF mutants (truncating, splice-site) may matter for emerging mutant-p53 reactivator drugs (eprenetapopt) where residual fold is required — R282W generally considered reactivatable in early-phase trials.

Used By

Biomarker