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JAK2 mutation (V617F or exon 12)

Deterministic view of the source YAML entity. Clinical authority remains with the cited source IDs and reviewer sign-off state.

IDBIO-JAK2
TypeBiomarker
Aliases
JAK2 V617FJAK2 exon 12JAK2 mutationМутація JAK2 (V617F або екзон 12)
Statusreviewed 2026-04-27 | pending_clinical_signoff
DiseasesNone declared
SourcesSRC-ESMO-MPN-2015 SRC-NCCN-MPN-2025

Biomarker Facts

Biomarker typegene_mutation
Mutation details{"functional_impact": "Constitutive JAK2 kinase activation through JH2-domain release → JAK-STAT signaling", "gene": "JAK2", "gene_hugo_id": "HGNC:6192", "hotspots": ["V617F (exon 14, JH2 pseudokinase domain — ~95% of PV; ~50–60% of ET / PMF)", "exon 12 (small in-frame indels — ~3% of PV; JAK2-V617F-negative PV)"], "variant_type": "missense / in-frame indel"}
Measurement
MethodAllele-specific PCR (V617F screen — high sensitivity LoD ~0.1% VAF) + Sanger / NGS for exon 12 if V617F-negative
Unitscategorical (positive/negative); VAF reported (allele burden tracks disease)
Sensitivity requirementASP / ddPCR LoD ~0.1% VAF for low-burden detection; allele burden useful for treatment monitoring
Actionability lookup{"gene": "JAK2", "variant": "V617F"}
Related biomarkersBIO-CALR

Notes

Diagnostic for PV (V617F or exon 12 — together ≥98%), ET (~50–60% V617F), PMF (~50–60% V617F). Mutually exclusive with CALR and MPL. JAK2 inhibitors (ruxolitinib COMFORT-I/II; fedratinib JAKARTA-2; pacritinib PERSIST-2; momelotinib MOMENTUM) treat MF symptoms + splenomegaly across all driver genotypes. Ruxolitinib is also 1L for hydroxyurea-resistant / -intolerant PV (RESPONSE) and approved for symptomatic / high-risk ET in some jurisdictions. Allele burden reduction on therapy correlates with longer-term outcomes in PV / MF but is not a treatment switch trigger.

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