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PDGFRA D842V in GIST is imatinib-RESISTANT (distinct from imatinib-sensitive PDGFRA non-D...

Deterministic view of the source YAML entity. Clinical authority remains with the cited source IDs and reviewer sign-off state.

IDBMA-PDGFRA-D842V-GIST
TypeActionability
Statusreviewed 2026-04-27 | pending_clinical_signoff | actionability review required
DiseasesDIS-GIST
SourcesSRC-CIVIC SRC-FDA-CDS-2026 SRC-NCCN-MELANOMA-2025

Actionability Facts

BiomarkerBIO-PDGFRA
VariantD842V (activation loop, exon 18 — ~7% of GIST, predominantly gastric)
DiseaseDIS-GIST
ESCAT tierIA
Recommended combinationsavapritinib monotherapy (1L for advanced D842V GIST — irrespective of line)
Contraindicated monotherapyimatinib (D842V resistant), sunitinib (D842V resistant), regorafenib (D842V resistant), ripretinib (modest activity in D842V; reserve for avapritinib failure)
Evidence summaryPDGFRA D842V in GIST is imatinib-RESISTANT (distinct from imatinib-sensitive PDGFRA non-D842V mutations). Avapritinib (NAVIGATOR phase 1/2 + VOYAGER phase 3, Heinrich Lancet Oncol 2020 / Kang JCO 2022 — ORR 88-91% in D842V GIST; treatment-naive cohort) is FDA-approved for advanced D842V GIST. Imatinib, sunitinib, regorafenib are ineffective.

Notes

ESCAT IA. OncoKB Level 1. Source-gap: SRC-NCCN-SARCOMA / SRC-NCCN-GIST-2025 / SRC-NAVIGATOR / SRC-VOYAGER not yet ingested. CNS toxicity (cognitive effects, intracranial hemorrhage) is on-target / dose- related — dose modifications standard. Companion-diagnostic genotyping via NGS panel mandatory before avapritinib initiation. Adjuvant imatinib has NO benefit in D842V GIST — these patients should be observed post-resection.

Used By

No reverse references found in the YAML corpus.